Much of this potential damage is caused by elements in your body called free radicals. Free radicals react negatively with important organic substances like fatty acids, proteins and even DNA. The curcumin found in turmeric is a potent antioxidant that helps neutralize these free radicals, preventing cellular damage and degeneration.

Turmeric is also linked to reducing oxidative stress on vascular tissues that can increase risk of chronic disease. Turmeric may reverse steps in the heart disease process by improving the function of the lining of your blood vessels.

Hence, this helps regulate your blood pressure, blood clotting and other factors vital to heart health. Also, preliminary studies show that curcumin may reduce the number of heart attacks bypass patients had after surgery. Turmeric has been proven to help lower brain disease risk.

Evidence shows that curcumin may affect brain function and the development of dementia. Furthermore, curcumin may be effective in delaying or reversing brain diseases and age-related decreases in brain function. This happens by triggering a growth hormone in the brain that helps spur new brain cell growth.

And, there are early signs that turmeric can even improve your memory. Turmeric has long been used to help soothe stomach pain, relieve constipation and control irritable bowel syndrome.

More so, its anti-inflammatory properties can lower your chance of developing ulcers and remove digestive system irritation. You can generally find turmeric in the spice aisle of your local grocery store.

Turmeric, when taken orally or applied to the skin, is considered safe for health purposes. Taking turmeric with black pepper enhances absorption.

Before taking turmeric or changing your diet, we recommend reaching out to a doctor. Visit mercy. com to find a physician near you today.

For other ways to fuel your health, check out foods full of heart-healthy fats. As always, you should consult your doctor before making any major changes to your diet.

com or call to find a doctor near you today. Healthy Living Top 5 Health Benefits of Turmeric Jan 22 Anti-Inflammatory Properties If you want to control inflammation, turmeric can do the trick. Antioxidants Oxidative damage is thought to be a trigger for aging and a variety of diseases.

Lowered Risk of Heart Disease Turmeric may reverse steps in the heart disease process by improving the function of the lining of your blood vessels. Prevention of Brain Disease Turmeric has been proven to help lower brain disease risk.

Increased Digestive Health Turmeric has long been used to help soothe stomach pain, relieve constipation and control irritable bowel syndrome. Where can you find turmeric? Related Posts Sore Throat vs. When to Keep a Sick Kid Home from School. Can You Get a Pap Smear on Your Period? Firestein Answers This Question and More.

Sue I have A-fib and take Xarelto. Is it safe for me to take turmeric? January 23rd, am. Lynda How much turmeric SHOUKD be taken daily? January 25th, pm. Curcumin is also a powerful antioxidant. Antioxidants scavenge molecules in the body known as free radicals, which damage cell membranes, tamper with DNA, and even cause cell death.

Antioxidants can fight free radicals and may reduce or even help prevent some of the damage they cause. In addition, curcumin lowers the levels of two enzymes in the body that cause inflammation. It also stops platelets from clumping together to form blood clots.

Curcumin stimulates the gallbladder to produce bile, which some people think may help improve digestion. The German Commission E, which determines which herbs can be safely prescribed in Germany, has approved turmeric for digestive problems.

And one double-blind, placebo-controlled study found that turmeric reduced symptoms of bloating and gas in people suffering from indigestion. Turmeric may help people with ulcerative colitis stay in remission.

Ulcerative colitis is a chronic disease of the digestive tract where symptoms tend to come and go. In one double-blind, placebo-controlled study, people whose ulcerative colitis was in remission took either curcumin or placebo, along with conventional medical treatment, for 6 months.

Those who took curcumin had a significantly lower relapse rate than those who took placebo. Turmeric does not seem to help treat stomach ulcers. In fact, there is some evidence that it may increase stomach acid, making existing ulcers worse.

See "Precautions" section. Because of turmeric's ability to reduce inflammation, researchers have wondered if it may help relieve osteoarthritis pain.

One study found that people using an Ayurvedic formula of herbs and minerals with turmeric, winter cherry Withinia somnifera , boswellia Boswellia serrata , and zinc had less pain and disability. But it's impossible to know whether turmeric, one of the other supplements, or all of them together, was responsible for the effects.

Early studies suggested that turmeric may help prevent atherosclerosis, the buildup of plaque that can block arteries and lead to heart attack or stroke. In animal studies, an extract of turmeric lowered cholesterol levels and kept LDL bad cholesterol from building up in blood vessels.

Because it stops platelets from clumping together, turmeric may also prevent blood clots from building up along the walls of arteries. But a double-blind, placebo-controlled study found that taking curcumin, the active ingredient in turmeric, at a dose of up to 4 g per day did not improve cholesterol levels.

There has been a great deal of research on turmeric's anti-cancer properties, but results are still preliminary. Evidence from test tube and animal studies suggests that curcumin may help prevent or treat several types of cancers, including prostate, breast, skin, and colon cancer.

Tumeric's preventive effects may relate to its antioxidant properties, which protect cells from damage. More research is needed. Cancer should be treated with conventional medications. Don't use alternative therapies alone to treat cancer. If you choose to use complementary therapies along with your cancer treatment, make sure you tell all your doctors.

Test tube and animal studies suggest turmeric may kill bacteria and viruses, but researchers don't know whether it would work in people. A preliminary study suggests curcumin may help treat uveitis, an inflammation of the eye's iris.

Preliminary research suggests that curcumin may be as effective as corticosteroids, the type of medication usually prescribed. Tumeric's powerful antioxidant, anti-inflammatory, and circulatory effects may help prevent and treat neurodegenerative diseases, including Alzheimer disease, Parkinson's disease, multiple sclerosis, and other conditions.

A relative of ginger, turmeric is a perennial plant that grows 5 to 6 feet high in the tropical regions of Southern Asia, with trumpet-shaped, dull yellow flowers. Its roots are bulbs that also produce rhizomes, which then produce stems and roots for new plants.

Turmeric is fragrant and has a bitter, somewhat sharp taste. Although it grows in many tropical locations, the majority of turmeric is grown in India, where it is used as a main ingredient in curry. The roots, or rhizomes and bulbs, are used in medicine and food.

They are generally boiled and then dried, turning into the familiar yellow powder. Curcumin, the active ingredient, has antioxidant properties. Other substances in this herb have antioxidant properties as well. Bromelain increases the absorption and anti-inflammatory effects of curcumin, so it is often combined with turmeric products.

Pediatric Turmeric supplements haven't been studied in children, so there is no recommended dose. The use of herbs is a time-honored approach to strengthening the body and treating disease.

However, herbs can trigger side effects and may interact with other herbs, supplements, or medications. For these reasons, you should take herbs with care, under the supervision of a health care provider.

Turmeric in food is considered safe. However, taking large amounts of turmeric and curcumin in supplement form for long periods of time may cause stomach upset and, in extreme cases, ulcers. People who have gallstones or obstruction of the bile passages should talk to their doctor before taking turmeric.

If you have diabetes, talk to your doctor before taking turmeric supplements. Turmeric may lower blood sugar levels. When combined with medications for diabetes, turmeric could cause hypoglycemia low blood sugar.

Although it is safe to eat foods with turmeric, pregnant and breastfeeding women should not take turmeric supplements. Because turmeric may act like a blood thinner, you should stop taking it at least 2 weeks before surgery.

Tell your doctor and surgeon that you have been taking turmeric. If you are being treated with any of the following medications, you should not use turmeric or curcumin in medicinal forms without first talking to your health care provider.

Turmeric may strengthen the effects of these drugs, raising the risk of bleeding. Blood thinners include warfarin Coumadin , clopidogrel Plavix , and aspirin, among others. Turmeric may interfere with the action of these drugs, increasing the production of stomach acid:. Turmeric may strengthen the effects of these drugs, increasing the risk of hypoglycemia low blood sugar.

Aggarwal BB, Yuan W, Li S, Gupta SC. Curcumin-free tumeric exhibits anti-inflammatory and anticancer activities: Identification of novel components of tumeric. Mol Nutr Food Res.

Aggarwal BB, Sundaram C, Malani N, Ichikawa H. Curcumin: the Indian solid gold. Adv Exp Med Biol. Asai A, Miyazawa T. Dietary curcuminoids prevent high-fat diet-induced lipid accumulation in rat liver and epididymal adipose tissue.

J Nutr. Asher GN, Spelman K. Clinical utility of curcumin extract. Altern Ther Health Med. Baum L, Cheung SK, Mok VC, et al. Curcumin effects on blood lipid profile in a 6-month human study. Pharmacol Res. Blumenthal M, Goldberg A, Brinckmann J. Herbal Medicine: Expanded Commission E Monographs.

Newton, MA: Integrative Medicine Communications; Bolognia JL, Jorizzo JL, Schaffer JV, eds. Philadelphia, PA: Elsevier Saunders; Darvesh AS, Aggarwal BB, Bishayee A. Curcumin and Liver Cancer: A Review. Curr Pharm Biotechnol.

Davis JM, Murphy EA, Carmichael MD, et al. Curcumin effects on inflammation and performance recovery following eccentric exercise-induced muscle damage. Am J Physiol Regul Integr Comp Physiol. Dorai T, Cao YC, Dorai B, Buttyan R, Katz AE. Therapeutic potential of curcumin in human prostate cancer.

Curcumin inhibits proliferation, induces apoptosis, and inhibits angiogenesis of LNCaP prostate cancer cells in vivo.

Dorai T, Gehani N, Katz A. Curcumin inhibits tyrosine kinase activity of epidermal growth factor receptor and depletes the protein. Mol Urol. Funk JL, Frye JB, Oyarzo JN, et al. Efficacy and mechanism of action of turmeric supplements in the treatment of experimental arthritis. Arthritis Rheum.

Gautam SC, Gao X, Dulchavsky S. Immunodilation by curcumin. Gescher AJ, Sharma RA, Steward WP.

Ulus Travma Acil Cerrahi Derg ; 26 : — Klingbeil AU , Schneider M , Martus P , Messerli FH , Schmieder RE. A meta-analysis of the effects of treatment on left ventricular mass in essential hypertension. Rasyid A , Lelo A. The effect of curcumin and placebo on human gall-bladder function: an ultrasound study.

Aliment Pharmacol Ther ; 13 : — Oxford University Press is a department of the University of Oxford. It furthers the University's objective of excellence in research, scholarship, and education by publishing worldwide.

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Article Contents Abstract. Lead author biography. Data Availability. Supplementary material. Journal Article. Effects of high-absorption curcumin for the prevention of hypertensive heart disease: a double-blind, placebo-controlled, randomized clinical study.

Masafumi Funamoto , Masafumi Funamoto. Division of Translational Research, Clinical Research Institute, National Hospital Organization Kyoto Medical Center.

Division of Molecular Medicine, School of Pharmaceutical Sciences, University of Shizuoka. Department of Pharmacology, Institute of Biomedical Sciences, Tokushima University Graduate School.

Oxford Academic. Yoichi Sunagawa. Yasufumi Katanasaka. Toru Kato. Department of Clinical Research, National Hospital Organization Tochigi Medical Center.

Junichi Funada. Department of Cardiology, National Hospital Organization Ehime Medical Center. Yoichi Ajiro. Division of Clinical Research, National Hospital Organization Yokohama Medical Center.

Maki Komiyama. Masaharu Akao. Akihiro Yasoda. Hajime Yamakage. Noriko Satoh-Asahara , Noriko Satoh-Asahara. Hiromichi Wada.

Yasumasa Ikeda. Tatsuya Morimoto. Koji Hasegawa. Corresponding author. Revision received:. PDF Split View Views. Select Format Select format. ris Mendeley, Papers, Zotero. enw EndNote. bibtex BibTex. txt Medlars, RefWorks Download citation.

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Open in new tab Download slide. Curcumin , Hypertension , Cardiac hypertrophy , Diastolic dysfunction , B-type natriuretic peptide , Prevention. Figure 1. Table 1 Baseline characteristics of the participants in the placebo and curcumin groups. P -value. Open in new tab. Table 2 Per cent changes of parameter in each group.

Mean difference. Figure 2. Table 3 Per cent change in brain natriuretic peptide divided by baseline value in each group. P -value interaction. Google Scholar Crossref. Search ADS. Google Scholar PubMed. OpenURL Placeholder Text. Published by Oxford University Press on behalf of the European Society of Cardiology.

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Turmeric Curcuma longa , an Indian spice, is a yellow pigment that is used worldwide in cooking, cosmetics, dyes, and medicines [ 8 ]. It is worth noting that turmeric is a frequently used food additive in Southeast Asia, which improves color and flavor of food preparations.

Curcumin chemical name: diferuloylmethane is an active component of turmeric [ 9 ], which has the capacity to interact with hundreds of molecular targets. Several studies have demonstrated the protective effects of curcumin against many chronic diseases, including various cancers, pulmonary disorders, and autoimmune diseases [ 10 ].

It has been shown to attenuate oxidative stress [ 11 ] and to exert a cardioprotective effect owing to its lipid-lowering properties [ 12 , 13 , 14 ]. However, some contradictory results have also been reported [ 15 , 16 , 17 , 18 , 19 , 20 , 21 ].

In a meta-analysis of 5 randomized controlled trials, overall effects in the entire study population as well as those in a subgroup analysis of subjects with cardiovascular risk factors failed to demonstrate a significant lipid-lowering effect of curcumin [ 22 ].

However, the studies included in this meta-analysis had limited sample sizes. Further, most of the randomized controlled trials that have reported positive effects of curcumin on blood lipid levels were published subsequent to the above-mentioned meta-analysis [ 23 , 24 , 25 , 26 , 27 , 28 ]. Nonetheless, conflicting reports exist in that some studies have reported promising effects [ 23 , 24 , 25 , 26 , 27 , 28 ], whereas others failed to demonstrate any significant effect [ 15 ].

Thus, we conducted a meta-analysis of published clinical trials to assess the efficacy and safety of turmeric and cucumin in lowering lipid levels in patients with risk factors for CVD. An online search was carried out for clinical studies published in English language in the following electronic databases: PubMed, Embase, Ovid, Medline and Cochrane Library.

All studies published as of November were eligible for inclusion. Medical Subject Heading MeSH terms were used for PubMed, and comparable terms were used for other databases. In addition, we used various synonyms. Primary outcome measures included serum levels of LDL-C, high-density lipoprotein cholesterol HDL-C , TG, and total cholesterol TC.

Treatment safety, measured by adverse effects due to turmeric and curcumin, was defined as a secondary outcome. Two independent investigators SQ and LFH screened the titles and abstracts of articles initially retrieved on online search of databases, and extracted essential data from eligible full-text articles.

Data on study design, patient characteristics, number of patients, treatment regimen, duration of treatment, year of publication, and daily dose of curcumin or turmeric powder were extracted, as were the mean ± SD or mean ± SE of all efficacy measures specified earlier.

To control relative heterogeneity, only data of efficacy measures pertaining to the study period between 4 weeks and 3 months were extracted. For studies with missing data, authors were sent emails requesting details of these data. Risk of bias was assessed independently by 2 reviewers SQ and LFH using the Cochrane Handbook for Systematic Reviews of Interventions [ 30 ].

This tool allows for assessment of the study quality with respect to 6 domains: random sequence generation, allocation concealment, blinding, incomplete outcome data, selective reporting, and other bias. For each domain, the risk of bias was marked as low, unclear, or high. The meta-analysis was conducted by using Stata version In this analysis, only continuous variables were extracted.

With use of relevant formulae [ 32 ], these values were also calculated from medians and ranges. Statistical heterogeneity between trials was detected by the Chi-squared and I -square I 2 tests. Subgroup analyses were conducted to explore heterogeneity among studies with respect to underlying disease and form of intervention turmeric or curcumin used.

As only a single study had reported efficacy measures disaggregated by gender [ 27 ], subgroup analyses by gender could not be undertaken.

Of the articles retrieved on initial literature search, a detailed evaluation of 11 full-text articles resulted in the elimination of 2 studies [ 33 , 34 ], due to the use of the same patient groups, and another 2 studies [ 35 , 36 ], owing to incomplete data Fig.

Consequently, 7 randomized controlled trials RCTs , including a total of subjects, met the inclusion criteria and were selected for a qualitative analysis.

Basic characteristics of the included RCTs are summarized in Table 1 , and the pre- and post-intervention serum lipid parameters are presented in Additional file 1 : Table S1.

Of the 7 eligible studies, only studies that enrolled patients with T2DM [ 15 , 24 , 25 , 28 ] or MetS [ 23 , 26 , 27 ] were included. All studies were conducted in a double-blind manner with the exception of the trials by Selvi et al. Two of the studies were performed in Iran [ 23 , 24 ] and 2 [ 15 , 25 ] were conducted in India.

The remaining studies were conducted in Pakistan [ 26 ], Taiwan [ 27 ], and Thailand [ 28 ], respectively. The duration of these studies ranged from 4 weeks to 6 months. Specified outcomes were reported from all studies, with the exception of the study by Chuengsamarn et al.

The form of intervention and serum lipid parameters of studies at baseline and after intervention differed. The risk of bias in the individual studies is shown in Additional file 2 : Table S2.

Overall, these selected studies varied in terms of quality: of the 7 RCTs, 4 were classified as high quality [ 24 , 26 , 27 , 28 ] and 3 were judged to be of moderate quality [ 15 , 23 , 25 ].

Four studies used appropriate randomization methods, such as a random number table [ 25 , 27 ] or a computer-generated list of random numbers [ 24 , 28 ]. Allocation concealment was only used in 4 studies [ 23 , 25 , 26 , 28 ]. Five trials used double-blinding of patients and practitioners [ 23 , 24 , 26 , 27 , 28 ].

All studies reported dropout rates and specific reasons for dropout with the exception of the trial by Chuengsamarn et al.

Forest plot of the meta-analysis for comparison of plasma LDL-C concentrations between experimental and control groups. Forest plot of the meta-analysis for comparison of plasma HDL-C concentrations between experimental and control groups. Forest plot of the meta-analysis for comparison of plasma TG concentrations between experimental and control groups.

Forest plot of the meta-analysis for comparison of plasma TC concentrations between experimental and control groups. To assess sources of potential bias, we conducted subgroup analyses by underlying diseases and forms of intervention—namely, studies in patients with hyperglycemia pre-diabetes and T2DM and MetS Table 2.

Five RCTs reported adverse effects in both the control and experimental groups. Rahmani et al. Amin et al. Chuengsamarn et al. Moreover, 4 patients experienced side effects in the placebo group: vertigo and itching, constipation, and hot flashes in 1 patient each. Selvi et al. None of the remaining studies reported any adverse reactions of turmeric and curcumin therapy.

No serious adverse reaction induced by turmeric and curcumin was reported in any of the studies included in this meta-analysis. The epidemic of obesity has contributed to a growing burden of CVD risk factors such as T2DM and MetS [ 37 ] defined as the presence of at least 3 out of the 4 criteria: central obesity, increased blood pressure, high blood sugar levels, and dyslipidemia [ 38 ].

Dyslipidemia is a well-established modifiable cardiovascular risk factor. All of the currently available antilipemic therapies have their own inherent shortcomings and disadvantages.

Therefore, natural treatments have been investigated as potential therapies for lowering blood lipid levels. This systematic review of 7 randomized trials of turmeric and curcumin in patients at risk of CVD identified evidence of their beneficial effects on serum TG and LDL-C levels, although no significant difference was found with respect to serum HDL levels.

When the analysis was restricted to more homogenous studies based on underlying disease in subjects hyperglycemia and MetS , a beneficial effect of turmeric and curcumin on serum TC levels was observed in subjects with MetS; however, in subjects with hyperglycemia, this beneficial effect on serum TC levels was not observed.

It seems that the natural form turmeric and curcumin have more positive effects on patients suffering from MetS. With regard to the forms of intervention, turmeric extract may have a greater beneficial effect on serum TC levels, as compared to that of turmeric in its natural form.

However, owing to the limited number of studies, definitive conclusions may not be drawn in this respect. Furthermore, larger scale trials are required among patients with MetS to explore the effect of turmeric extract, even in novel forms, in lowering plasma TC concentrations.

Sahebkar conducted a meta-analysis of 5 RCTs to assess the effects of curcumin on blood lipid levels and found no significant improvements in the lipid profile in any aspect [ 22 ].

Several explanations could be tendered to explain why the results of their study were contrary to those of the present study. Firstly, both parallel and crossover randomized trials were selected, and these may have adversely influenced the final results.

Secondly, most of the selected studies were conducted with unformulated curcumin, which is considered to have low bioavailability. Curcumin has poor bioavailability owing to its poor absorption, fast metabolism, and rapid elimination from the body.

Some attempts have been made to overcome these deficits, including the use of a piperine black pepper adjuvant, liposomal curcumin, nanoparticles, phospholipid complexes, and an amorphous form [ 39 , 40 ].

Therefore, a hypothesis could be proposed that these novel dosage forms of curcumin may achieve greater clinical effects. To verify this theory, forest plots were conducted initially. Nevertheless, in our present review, only two trials [ 23 , 24 ] used novel forms, and the preparations used in these studies were dissimilar amorphous forms were used in one study [ 23 ] and nanoparticles were used in the other [ 24 ].

Therefore, further research on newer dosage forms of curcumin is required to confirm the hypothesis. Finally, differences with respect to underlying diseases in the study population may also explain this discrepancy. The authors included healthy participants and patients with various chronic diseases i.

Even on performing a subgroup analysis in patients with high cardiovascular risk acute coronary syndrome, T2DM, or concomitant dyslipidemia and obesity , no significant difference could be identified because coronary artery disease is an end event rather than a risk factor for CVD.

The article by Mohammadi et al. Unlike this study, in a trial among patients with coronary artery disease, although curcumin supplementation decreased serum levels of TC, LDL-C, and TG, there was no obvious difference when compared to placebo [ 41 ], possibly due to the small size of the study.

Subsequently, a study by Soare et al. found that mg of curcumin did not influence plasma lipid levels in non-obese relatively healthy individuals [ 42 ]. Therefore, we tentatively propose that the antilipemic effect of curcumin is evident only in patients who are at a higher risk of cardiovascular morbidity, such as those with MetS, T2DM, and obesity.

Some molecular mechanisms could potentially explain these results. Insulin resistance IR is the basic underlying pathology in both T2DM and MetS. Neerati et al. reported that curcumin could counter IR [ 43 ].

Through amelioration of metabolic derangement and potential binding of curcumin with peroxisome proliferator-activated receptor gamma PPAR-γ as agonist, curcumin could play a preventive role in diet-induced insulin resistance [ 44 ].

Moreover, curcumin was shown to increase activation of PPAR-γ [ 45 ], which suppressed expression of the LDL-C receptor gene, and could thereby reduce plasma LDL-C concentrations [ 46 ].

Because it interacts with multiple targets, including peroxisome proliferator-activated receptor alpha PPAR-α , PPAR-γ, cholesteryl ester transfer protein CETP , and lipoprotein lipase, curcumin could probably play a role in reduction of triglyceride levels [ 47 , 48 , 49 ].

Furthermore, curcumin is expected to affect both synthesis and catabolism of triglyceride-rich lipoproteins [ 47 , 48 , 49 ]. Thus, curcumin supplementation may lower plasma triglycerides and cholesterol concentrations by mitigating the expressions of lipogenic genes [ 48 , 49 , 50 ].

Additionally, the lipid-lowering effect of turmeric and curcumin is related to statins. Panahi et al. found that curcumin affected all pathways of cholesterol metabolism that are affected by statin therapy; it also reduced the effective doses of statins, which helped reduce the incidence of serious adverse reactions [ 51 ].

Furthermore, curcumin might serve as a valuable adjunct to statin therapy in patients with disordered lipid metabolism [ 51 ]. In our meta-analysis, we found that consumption of turmeric and curcumin was safe and well-tolerated in general. Several potential limitations of this review need mention.

First, the most important limitation may pertain to the interpretability of outcomes. Third, all subjects in the included studies were Asians. Lastly, some data were obtained indirectly, and those could have affected the accuracy of both the overall effects and the results of subgroup analyses.

Subjects who received turmeric and curcumin experienced a natural cardioprotective effect, with lowering of serum LDL-C and TG levels, as compared to subjects who did not. The efficacy of turmeric and curcumin on serum TC levels remains inconclusive, despite their superior efficacy observed in patients with MetS.

A greater effect of turmeric extract in reducing serum TC levels may be observed in patients who are at risk of CVD; however, this finding needs to be confirmed in future studies. No significant change in serum HDL levels was observed.

Due to uncertainties related to dosage form, dose and medication frequency, it is premature to recommend the use of turmeric or curcumin in clinical settings. Nonetheless, the analysis does provide a synthesis of the currently available evidence and supports larger scale clinical trials of curcumin.

World Health Organization: The Top 10 Causes of Death. html Accessed 20 Nov Srikanth S, Deedwania P. Management of Dyslipidemia in patients with hypertension, diabetes, and metabolic syndrome. Curr Hypertens Rep. Article PubMed Google Scholar. Musunuru K. Atherogenic dyslipidemia: cardiovascular risk and dietary intervention.

Article CAS PubMed PubMed Central Google Scholar. Padala S, Thompson PD. Statins as a possible cause of inflammatory and necrotizing myopathies. Article CAS PubMed Google Scholar. Chalasani N. Statins and hepatotoxicity: focus on patients with fatty liver. Sniderman AD.

Is there value in liver function test and creatine phosphokinase monitoring with statin use? Am J Cardiol. Law M, Rudnicka AR. Statin safety: a systematic review. Turmeric has been proven to help lower brain disease risk. Evidence shows that curcumin may affect brain function and the development of dementia.

Furthermore, curcumin may be effective in delaying or reversing brain diseases and age-related decreases in brain function. This happens by triggering a growth hormone in the brain that helps spur new brain cell growth. And, there are early signs that turmeric can even improve your memory.

Turmeric has long been used to help soothe stomach pain, relieve constipation and control irritable bowel syndrome. More so, its anti-inflammatory properties can lower your chance of developing ulcers and remove digestive system irritation. You can generally find turmeric in the spice aisle of your local grocery store.

Turmeric, when taken orally or applied to the skin, is considered safe for health purposes. Taking turmeric with black pepper enhances absorption. Before taking turmeric or changing your diet, we recommend reaching out to a doctor. Visit mercy.

com to find a physician near you today. For other ways to fuel your health, check out foods full of heart-healthy fats. As always, you should consult your doctor before making any major changes to your diet.

com or call to find a doctor near you today. Healthy Living Top 5 Health Benefits of Turmeric Jan 22 Anti-Inflammatory Properties If you want to control inflammation, turmeric can do the trick. Antioxidants Oxidative damage is thought to be a trigger for aging and a variety of diseases.

Lowered Risk of Heart Disease Turmeric may reverse steps in the heart disease process by improving the function of the lining of your blood vessels. Prevention of Brain Disease Turmeric has been proven to help lower brain disease risk.

Increased Digestive Health Turmeric has long been used to help soothe stomach pain, relieve constipation and control irritable bowel syndrome. Where can you find turmeric? Related Posts Sore Throat vs. When to Keep a Sick Kid Home from School.

Can You Get a Pap Smear on Your Period?